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UnDerstAnDing sPeciFic heArt DiseAse MeDicAtions ACE angiotensin converting enzyme ; inhibitors are medications that widen or dilate your blood vessels to improve the amount of blood your heart pumps and lower blood pressure, helping to decrease the amount of work your heart has to do. Examples of ACE inhibitors include Capoten, Vasotec, Prinivil, Zestril, Lotensin, and Monopril, Altace, Accupril, Aceon, Mavik, and Univasc. Diuretics -- "water pills" -- help your body get rid of unneeded water and salt through the urine. Getting rid of excess. 149. TUREK, B.; KODL, J.: 1.2.2 Cizorod ltky a aditiva. In: Manul prevence v lkask praxi. II. Vziva. Praha, Sttn zdravotn stav 1995, S. 34-38. [autor kapitoly, ucebnice VS]. 150. VOJTCHOVSK, M.: Geriatrick psychiatrie II. In: Francov, D.: Stbrn mozaika. 2.dl. Praha, Cesk rozhlas 1995, S. 66-85. [autor kapitoly, monografie]. 151. ANDL, M.: Diabetes mellitus. In: Andl, M.; Benes, P.; Dlouh, P.; Hampl, R.; Kalvachov, B.; Kraml, P.; Kuzela, L.; Mal, J.; Neradilov, M.; Nmec, J.; Pytlk, R.; Strka, L.; Stich, V.; Zamrazil, V.: Vnitn lkastv. V. Endokrinologie, diabetologie, poruchy metabolismu a vzivy. Praha, Karolinum 1996, S. 97-122. [autor kapitoly, ucebnice VS]. 152. ANDL, M.: Diabetick dieta v lcb DM 2. typu. In: Perusicov, J. et al.: Diabetes mellitus 2. typu. Praktick rukov. Praha, Galn 1996, S. 40-50. [autor kapitoly, monografie]. 153. ANDL, M.: Diabetick dieta v lcb DM 2. typu. In: Perusicov, J. et al.: Diabetes mellitus 2. typu. Praktick rukov. Praha, Galn 1996, S. 99-107. [autor kapitoly, monografie]. 154. ANDL, M.: Glykogenzy. In: Andl, M.; Benes, P.; Dlouh, P.; Hampl, R.; Kalvachov, B.; Kraml, P.; Kuzela, L.; Mal, J.; Neradilov, M.; Nmec, J.; Pytlk, R.; Strka, L.; Stich, V.; Zamrazil, V.: Vnitn lkastv. V. Endokrinologie, diabetologie, poruchy metabolismu a vzivy. Praha, Karolinum 1996, S. 126-128. [autor kapitoly, ucebnice VS]. 155. ANDL, M.: Hypoglykemie. In: Andl, M.; Benes, P.; Dlouh, P.; Hampl, R.; Kalvachov, B.; Kraml, P.; Kuzela, L.; Mal, J.; Neradilov, M.; Nmec, J.; Pytlk, R.; Strka, L.; Stich, V.; Zamrazil, V.: Vnitn lkastv. V. Endokrinologie, diabetologie, poruchy metabolismu a vzivy. Praha, Karolinum 1996, S. 122-125. [autor kapitoly, ucebnice VS]. 156. ANDL, M.: Poruchy vnitnho prosted. In: Andl, M.; Benes, P.; Dlouh, P.; Hampl, R.; Kalvachov, B.; Kraml, P.; Kuzela, L.; Mal, J.; Neradilov, M.; Nmec, J.; Pytlk, R.; Strka, L.; Stich, V.; Zamrazil, V.: Vnitn lkastv. V. Endokrinologie, diabetologie, poruchy metabolismu a vzivy. Praha, Karolinum 1996, S. 162-180. [autor kapitoly, ucebnice VS]. 157. BAKOS, K.; DVOK, P.; KUPKA, K.; PRADCOV, J.; SMAL, M.; STICH, V.; URBNEK, J.: Emisn tomografie. In: Nuklern medicna. Jilemnice, Gentiana 1996, S. 26-33. [autor kapitoly, monografie]. 158. BRTOV, J.: 2.1.3 Pasivn kouen. In: Manul prevence v lkask praxi. III. Prevence nepznivho psoben vlivu obytnho prosted na zdrav. Praha, Sttn zdravotn stav 1996, S. 32-35. [autor kapitoly, ucebnice VS]. 159. BEDN, M.; KOZK, K.; KUBN, M.; MENCKOV, E.: Tycky spatn barviteln podle Grama, ppadn acidorezistentn. In: Bedn, M.; Schindler, J.; Frakov, V.; Soucek, A.; Vvra, J.: Lkask mikrobiologie. Bakteriologie, virologie, parazitologie. Praha, Marvil 1996, S. 305-318. [autor kapitoly, ucebnice VS]. 160. BEDN, M.; SCHARFEN, J.: Nesporulujc anaerobn bakterie. In: Bedn, M.; Schindler, J.; Frakov, V.; Soucek, A.; Vvra, J.: Lkask mikrobiologie. Bakteriologie, virologie, parazitologie. Praha, Marvil 1996, S. 289-297. [autor kapitoly, ucebnice VS]. 161. BENES, J.: Lcba grampozitivm sepse. In: Sbornk pednsek - III. Zaludovy dny, st n. L., 28.-29.3.96. st n. L., b.v. 1996, nestr.5 s. [autor stat, sbornk]. 162. BENES, P.; ANDL, M.: Podvziva. In: Andl, M.; Benes, P.; Dlouh, P.; Hampl, R.; Kalvachov, B.; Kraml, P.; Kuzela, L.; Mal, J.; Neradilov, M.; Nmec, J.; Pytlk, R.; Strka, L.; Stich, V.; Zamrazil, V.: Vnitn lkastv. V. Endokrinologie, diabetologie, poruchy metabolismu a vzivy. Praha, Karolinum 1996, S. 148-149. [autor kapitoly, ucebnice VS]. 163. CIESLAROV, B.: Choroby jater a zlucovch cest. Klinick projevy jaternch chorob. In: Strejcek, J.; Havel, J.: Merck Manual. Kompendium klinick medicny. Praha, X-Egem 1996, S. 771-783. [autor kapitoly, monografie]. 164. CIESLAROV, B.: Choroby jater a zlucovch cest. Laboratorn a zaobrazovac vysetovac metody jater a zlucovch cest. In: Strejcek, J.; Havel, J.: Merck Manual. Kompendium klinick medicny. Praha, X-Egem 1996, S. 763-771. [autor kapitoly, monografie]. 165. CIESLAROV, B.: Choroby jater a zlucovch cest. vod. In: Strejcek, J.; Havel, J.: Merck Manual. Kompendium klinick medicny. Praha, X-Egem 1996, S. 762-763. [autor kapitoly, monografie]. 166. CELKO, A.M.: Antrax. In: Manul prevence v lkask praxi. IV. Zklady prevence infekcnho onemocnn. Praha, Sttn zdravotn stav 1996, S. 17-18. [autor kapitoly, ucebnice VS]. 167. CELKO, A.M.: Chlamydiov infekce. In: Manul prevence v lkask praxi. IV. Zklady prevence infekcnho onemocnn. Praha, Sttn zdravotn stav 1996, S. 54-58. [autor kapitoly, ucebnice VS]. 168. CELKO, A.M.: Infekcn mononukleza. In: Manul prevence v lkask praxi. IV. Zklady prevence infekcnho onemocnn. Praha, Sttn zdravotn stav 1996, S. 61-62. [autor kapitoly, ucebnice VS]. 169. CELKO, A.M.: Legionelza. In: Manul prevence v lkask praxi. IV. Zklady prevence infekcnho onemocnn. Praha, Sttn zdravotn stav 1996, S. 67. [autor kapitoly, ucebnice VS]. 170. CELKO, A.M.: Nestovice plan, psov opar. In: Manul prevence v lkask praxi. IV. Zklady prevence infekcnho onemocnn. Praha, Sttn zdravotn stav 1996, S. 84-85. [autor kapitoly, ucebnice VS]. 171. CELKO, A.M.: Pusnice. In: Manul prevence v lkask praxi. IV. Zklady prevence infekcnho onemocnn. Praha, Sttn zdravotn stav 1996, S. 93-94. [autor kapitoly, ucebnice VS]. 172. CELKO, A.M.: Spalnicky. In: Manul prevence v lkask praxi. IV. Zklady prevence infekcnho onemocnn. Praha, Sttn zdravotn stav 1996, S. 101-102. [autor kapitoly, ucebnice VS] and xenical.
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Krakow K, University of Frankfurt, Mainz, Germany Since the demonstration a decade ago that functional magnetic resonance imaging fMRI ; is able to provide high spatial resolution maps of brain function, considerable effort has been directed at applying this technique to study patients with epilepsy, in particular patients with intractable epilepsy considered for epilepsy surgery. Successful epilepsy surgery is vitally dependent on the accurate determination of the epileptic focus and of the risks of postoperative neurological deficits. First, through determination of eloquent cortical areas, fMRI may predict a deficit in cognitive e.g., language and memory ; or sensorimotor functions that might arise from surgical intervention. Second, asymmetries in the activation pattern may facilitate the lateralization and localization of a functional-deficit zone. For example, identification of functionally hypoactive temporal lobe structures in memory paradigms, which are normally known to be associated with symmetrical temporal lobe activation, may have predictive value for lateralization of seizure foci in temporal lobe epilepsy. Finally, fMRI can provide more direct evidence for the localization of epileptic foci through ictal or interictal blood oxygenation level dependent BOLD ; activation studies. It could be demonstrated that ictal fMRI is capable of imaging reversible BOLD signal changes associated with epileptic seizures, with the abnormality localized closely to the site of maximum electric abnormality. However, ictal fMRI is not routinely practicable for several reasons: Most ictal events are associated with head and body movement and impairment of consciousness, usually to a degree that the required level of cooperation for an MRI scan cannot be achieved. Furthermore, seizures occur usually unpredictably and are short lasting. It is therefore impracticable for a patient to lie in a MR scanner awaiting the onset of a seizure. For these practical issues, the investigation of ictal activity will be limited to highly selected patients who have very frequent seizures without gross head movement, or have epilepsy syndromes with seizures occurring predictably, e.g., reflex epilepsies. Compared with ictal fMRI, mapping of interictal epileptiform discharges IED ; has several advantages: 1 ; IED are a common phenomenon in patients with epilepsy; 2 ; IED are not associated with stimulus-correlated motion; and 3 ; fMRI activation associated with single discharges is less likely to be confounded with propagation effects compared with ongoing ictal activity. As IED are by definition a sub-clinical phenomenon, a second modality is necessary to identify these events. Hence, this approach was only made possible by recording EEG during fMRI EEG-correlated fMRI ; . However, the clinical interpretation of the fMRI maps is difficult; mainly because results of EEG-correlated fMRI have not been systematically validated with a gold standard, which are intracranial recordings and outcome after epilepsy surgery. Before EEG-correlated fMRI might be used as a decisive method in the presurgical assessment of epilepsy patients, the relation between fMRI results, invasive EEG recordings and surgical outcome in relation to resection of the activated area has to be established. Currently, EEG-correlated fMRI has to be considered a research tool providing insights to the pathophysiological processes underlying epileptic disorders. It remains unclear if the method will also be used for the routine clinical work-up of epilepsy patients in the future and zestoretic.
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